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Eating disorders and other factors - obesity PDF Print E-mail
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Eating disorders

Why certain individuals continue to eat despite obesity is now being questioned as a behavioural disorder. Other factors, which would determine eating patterns, depend on availability and attractiveness of food and physiological indicators of hunger and satiety. The emotional factor may determine these, and may trigger or modulate the control of food intake.

Chemicals like serotonin have emerged as key mediators determining food intake. These are known to affect the receptors in the hypothalamus in the brain, which primarily controls appetite. Based on the current information about these neurotransmitters, research is being aimed to evolve substances, which would influence the appetite centres. However till date, only a few drugs have been introduced in the market. Their usefulness and safety continue to be debatable. Most endocrinologists feel that these are rather desperate methods. Nevertheless a wide area of research is still open and what mediates hunger and satiety and why some achieve satiety with small amounts of food is a million-dollar question.

Genetics factors
Obesity often runs in families. This suggests a genetic predisposition, but at the same time common eating patterns, attitudes towards food cannot discount the environmental factors. Preventive measures should be initiated at early ages to prevent obesity in offsprings.

Endocrine obesity
A small percentage of the obese suffer from hormonal disorders. In such cases consultation with an endocrinologist may be warranted. In children, hypothalamic lesions i.e. non-functional tissues in the hypothalamus are commonly found. Craniophary-giomas i.e. a brain tumour derived from remnants of an embryonic structure, is sometimes present in children and the symptoms are obesity associated with growth retardation and visual abnormalities. Disorders of the thyroid gland leading to reduced functioning of the gland is also fairly common


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